Show simple item record

dc.contributor.authorF Motamedi
dc.contributor.authorA Rashidy-Pour
dc.contributor.authorM R Zarrindast
dc.contributor.authorM Badavi
dc.date.accessioned2017-10-24T12:17:51Z
dc.date.available2017-10-24T12:17:51Z
dc.identifier.citationrft.volume=110&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.spage=193&rft.issue=1-2&rft.epage=7&ctx_ver=Z39.88-2004&rft.jtitle=Psychopharmacology&rft.date=1993
dc.identifier.issn0033-3158
dc.identifier.urihttp://dsp.sbmu.ac.ir/xmlui/handle/123456789/80671
dc.description.abstractIntraperitoneal (IP) administration of bombesin (BBS; 2.5-20 micrograms/kg) induced a dose-dependent inhibition of food intake. The effect was decreased by intraventricular (ICV) administration of bombesin receptor antagonist [Leu14-psi (CH2NH)-Leu13] (3 micrograms/rat) but not by the D1 antagonist SCH 23390, the D2 antagonists sulpiride and pimozide, the dopamine antagonist cis-flupentixol, adrenoceptor blockers phenoxybenzamine or propranolol and serotonergic antagonist methergoline. It is concluded that BBS-induced suppression of feeding may be mediated through central BBS receptors, and is independent of interaction with brain catecholaminergic system.
dc.sourcePsychopharmacology
dc.titleBombesin-induced anorexia requires central bombesin receptor activation: independence from interaction with central catecholaminergic systems.


Files in this item

FilesSizeFormatView

There are no files associated with this item.

This item appears in the following Collection(s)

Show simple item record